Special AT-rich sequence-binding protein 2 (SATB2) expression is sensitive but may not be specific for osteosarcoma as compared with other high-grade primary bone sarcomas
Author:
Affiliation:
1. Department of Pathology and Laboratories; Seattle Children's Hospital/University of Washington; Seattle WA USA
2. Department of Anatomic Pathology; University of California; San Francisco CA USA
Funder
UCSF Department of Pathology
Publisher
Wiley
Subject
General Medicine,Histology,Pathology and Forensic Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/his.12911/fullpdf
Reference32 articles.
1. A cross-species analysis of SATB2 expression suggests deep conservation across vertebrate lineages;Sheehan-Rhooney;Dev. Dyn.,2010
2. A network connecting Runx2, SATB2, and the miR-23a~27a~24-2 cluster regulates the osteoblast differentiation program;Hassan;Proc. Natl Acad. Sci. USA,2010
3. SATB2 is a multifunctional determinant of craniofacial patterning and osteoblast differentiation;Dobreva;Cell,2006
4. Identification of SATB2 as the cleft palate gene on 2q32-q33;FitzPatrick;Hum. Mol. Genet.,2003
5. Heterozygous nonsense mutation SATB2 associated with cleft palate, osteoporosis, and cognitive defects;Leoyklang;Hum. Mutat.,2007
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