Tissue expression of aquaporin 3 in different sites of vitiligo: an immunohistochemical study
Author:
Affiliation:
1. Department of Dermatology; Faculty of Medicine; Cairo University; Cairo Egypt
2. Department of Pathology; Faculty of Medicine; Zagazig University; Zagazig Egypt
Publisher
Wiley
Subject
Infectious Diseases,Dermatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/jdv.15049/fullpdf
Reference10 articles.
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2. Melanocyte detachment after skin friction in non lesional skin of patients with generalized vitiligo;Gauthier;Br J Dermatol,2003
3. Acral lesions of vitiligo: why are they resistant to photochemotherapy?;Esmat;J Eur Acad Dermatol Venereol,2011
4. Matrix metalloproteinase in acral and non-acral vitiligo;Esmat;Photodermatol Photoimmunol Photomed,2017
5. Less keratinocyte-derived factors related to more keratinocyte apoptosis in depigmented than normally pigmented suction-blistered epidermis may cause passive melanocyte death in vitiligo;Lee;J Invest Dermatol,2005
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1. Aquaporins in Skin;Advances in Experimental Medicine and Biology;2023
2. Abnormal nuclear expression of aquaporin‐3 in lesional and perilesional skin of vitiligo patients: A novel immunohistochemical finding;Journal of Cosmetic Dermatology;2022-11-27
3. Downregulation of aquaporin 3 in patients with pemphigus vulgaris;Wound Repair and Regeneration;2022-05-05
4. Estimation of tissue level of human beta‐defensin 1 in vitiligo before and after narrowband ultraviolet B phototherapy: A case‐control study;Photodermatology, Photoimmunology & Photomedicine;2021-12-21
5. Human Aquaporins: Functional Diversity and Potential Roles in Infectious and Non-infectious Diseases;Frontiers in Genetics;2021-03-16
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