High levels of oncomi R ‐21 contribute to the senescence‐induced growth arrest in normal human cells and its knock‐down increases the replicative lifespan

Author:

Dellago Hanna1,Preschitz‐Kammerhofer Barbara1,Terlecki‐Zaniewicz Lucia1,Schreiner Carina1,Fortschegger Klaus2,Chang Martina W.‐F.1,Hackl Matthias1,Monteforte Rossella1,Kühnel Harald3,Schosserer Markus1,Gruber Florian4,Tschachler Erwin45,Scheideler Marcel6,Grillari‐Voglauer Regina178,Grillari Johannes17,Wieser Matthias18

Affiliation:

1. Department of Biotechnology BOKU‐VIBT University of Natural Resources and Life Sciences Vienna Muthgasse 181190Vienna Austria

2. CCRI ‐ Children's Cancer Research Institute Zimmermannplatz 101090 Vienna Austria

3. Institute of Physiology, Pathophysiology and Biophysics Department of Biomedical Sciences University of Veterinary Medicine Vienna Veterinärplatz 1A‐1210Vienna Austria

4. Department of Dermatology Medical University of Vienna A‐1090Vienna Austria

5. C.E.R.I.E.S. ‐ Centre de Recherches et d'Investigations Epidermiques et Sensorielles 20 Rue Victor Noir 92200 Neuilly‐sur‐Seine France

6. Institute for Genomics and Bioinformatics Graz University of Technology Petersgasse 148010Graz Austria

7. Evercyte GmbH Muthgasse 181190Vienna Austria

8. Austrian Centre of Industrial Biotechnology (ACIB GmbH) Muthgasse 181190Vienna Austria

Publisher

Wiley

Subject

Cell Biology,Ageing

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