Reduced amyloid‐β degradation in early A lzheimer's disease but not in the APP swe PS 1dE9 and 3x T g‐ AD mouse models-Reference-Cited by-同舟云学术

Reduced amyloid‐β degradation in early A lzheimer's disease but not in the APP swe PS 1dE9 and 3x T g‐ AD mouse models

Published:2013-04-23 Issue:3 Volume:12 Page:499-507
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Stargardt Anita¹,Gillis Judith¹,Kamphuis Willem²,Wiemhoefer Anne¹,Kooijman Lieneke²,Raspe Marcel¹,Benckhuijsen Willemien³,Drijfhout Jan W.³,M. Hol Elly²⁴,Reits Eric¹

Affiliation:

1. Department of Cell Biology and Histology Academic Medical Center Meibergdreef 151105 AZ Amsterdam The Netherlands

2. Department of Astrocyte Biology and Neurodegeneration Netherlands Institute for Neuroscience – an Institute of the Royal Netherlands Academy of Arts and Sciences (KNAW) Meibergdreef 47, 1105 BA Amsterdam The Netherlands

3. Department of Immunohematology and Blood Transfusion Leiden University Medical Center Albinusdreef 2, 2333 ZA Leiden The Netherlands

4. Swammerdam Institute for Life Sciences Center for Neuroscience University of Amsterdam Sciencepark 904 1098 XH AmsterdamThe Netherlands

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.12074

Reference50 articles.

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2. The Genetics of Alzheimer Disease: Back to the Future

3. Concerted changes in transcripts in the prefrontal cortex precede neuropathology in Alzheimer's disease

4. Neuropathological stageing of Alzheimer-related changes

5. Cytosolic and nuclear aggregation of the amyloid ?-peptide following its expression in the endoplasmic reticulum

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