Inhibition of MAPK ‐mediated ACE expression by compound C66 prevents STZ ‐induced diabetic nephropathy-Reference-Cited by-同舟云学术

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Inhibition of MAPK ‐mediated ACE expression by compound C66 prevents STZ ‐induced diabetic nephropathy

Published:2013-12-11 Issue:2 Volume:18 Page:231-241
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Pan Yong¹²,Huang Yi¹,Wang Zhe¹,Fang Qilu¹,Sun Yusheng³,Tong Chao¹,Peng Kesong¹,Wang Yangwei⁴,Miao Lining⁴,Cai Lu²,Zhao Yunjie¹,Liang Guang¹

Affiliation:

1. Chemical Biology Research Center School of Pharmaceutical Sciences Wenzhou Medical University Wenzhou Zhejiang China

2. Chinese‐American Research Institute for Diabetic Complications School of Pharmaceutical Sciences Wenzhou Medical University Wenzhou Zhejiang China

3. Department of General Surgery The First Affiliated Hospital of Wenzhou Medical University Wenzhou Zhejiang China

4. Department of Nephropathy the Second Hospital of Jilin University Changchun Jilin China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.12175

Reference40 articles.

1. Biochemistry and molecular cell biology of diabetic complications

2. The intrarenal renin–angiotensin system and diabetic nephropathy

3. Inhibition of intracellular Angiotensin II formation blocks high glucose effect on mesangial matrix

4. Early effects of high glucose in retinal tissue cultures

5. High glucose concentration stimulates intracellular renin activity and angiotensin II generation in rat mesangial cells

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