Omeprazole: inhibitor of both acid formation and translocation in gastric mucosa

Author:

EKBLAD E. B. M.

Publisher

Wiley

Subject

Physiology

Reference12 articles.

1. The sulfoxide moiety of substituted benzimidazoles is essential for inhibition of parietal cell K+/H+-ATPase;Beil;Br J Pharmacol,1986

2. Invariant relationship between total acid secretion and secreta-gogue exposure: secretory dynamics in bullfrog;Ekblad;Am J Physiol,1984

3. Thiocyanate and nitrite inhibit proton translocation in gastric mucosa;Ekblad;Biochim Biophys Acta,1985

4. Conservativc and nonconservative inhibitors of gastric acid secretion;Ekblad;Am J Physiol,1987

5. Substituted bcnzimidaxoles inhibit acid sccretion by blocking (H+-K+)-APTase;Fellenius;Nature,1981

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1. A Clinical Study of Post-treatment Nursing Strategy for Venous Malformations in the Pharynx Region;Iranian Red Crescent Medical Journal;2023-03-04

2. What dual-action agents reveal about acid secretion: A combined experimental and modeling analysis;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;1992-10

3. Dynamics of a metabolic system: what single-action agents reveal about acid secretion;American Journal of Physiology-Gastrointestinal and Liver Physiology;1992-03-01

4. Enhancement of antibiotic concentrations in gastric mucosa by H2-receptor antagonist;Digestive Diseases and Sciences;1991-01

5. Extracellular potassium is necessary for acid translocation but not for acid formation in gastric mucosa;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;1990-08

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