Z-ligustilide ameliorated ultraviolet B-induced oxidative stress and inflammatory cytokine production in human keratinocytes through upregulation of Nrf2/HO-1 and suppression of NF-κB pathway-Reference-Cited by-同舟云学术

Z-ligustilide ameliorated ultraviolet B-induced oxidative stress and inflammatory cytokine production in human keratinocytes through upregulation of Nrf2/HO-1 and suppression of NF-κB pathway

Published:2015-07-14 Issue:9 Volume:24 Page:703-708
ISSN:0906-6705
Container-title:Experimental Dermatology
language:en
Short-container-title:Exp Dermatol

Author:

Wu Zhouwei¹²,Uchi Hiroshi²,Morino-Koga Saori²,Shi Weimin¹,Furue Masutaka²

Affiliation:

1. Department of Dermatology; Shanghai First People's Hospital; Shanghai Jiaotong University; Shanghai China

2. Department of Dermatology; Graduate School of Medical Sciences; Kyushu University; Fukuoka Japan

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Dermatology,Molecular Biology,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/exd.12758/fullpdf

Reference44 articles.

1. Differential Activation of Signaling Pathways by UVA and UVB Radiation in Normal Human Epidermal Keratinocytes†

2. Environmental effects and skin disease

3. An integrated view of oxidative stress in aging: basic mechanisms, functional effects, and pathological considerations

4. Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain

5. Hypochlorous acid-induced heme oxygenase-1 gene expression promotes human endothelial cell survival

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