Deciphering the mechanism of calcineurin inhibitor‐induced hypertension
Author:
Affiliation:
1. Department of Cardiovascular and Renal Research, Institute of Molecular Medicine University of Southern Denmark Odense Denmark
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/apha.13976
Reference7 articles.
1. Activation of Kir4.1/Kir5.1 contributes to the cyclosporin A‐induced stimulation of the renal NaCl cotransporter and hyperkalemic hypertension;Gao ZX;Acta Physiol (Oxf),2023
2. The calcineurin inhibitor tacrolimus activates the renal sodium chloride cotransporter to cause hypertension
3. NaCl cotransporter abundance in urinary vesicles is increased by calcineurin inhibitors and predicts thiazide sensitivity
4. Inwardly rectifying K+ channels 4.1 and 5.1 (Kir4.1/Kir5.1) in the renal distal nephron
5. Potassium Modulates Electrolyte Balance and Blood Pressure through Effects on Distal Cell Voltage and Chloride
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1. Resveratrol attenuates cyclosporin A-induced upregulation of the thromboxane A2 receptor and hypertension via the AMPK/SIRT1 and MAPK/NF-κB pathways in the rat mesenteric artery;European Journal of Pharmacology;2024-06
2. Calcineurin inhibition, cardiovascular consequences, vascular resistance, and potential responses;Acta Physiologica;2024-01-12
3. Efficacy and safety of calcineurin inhibitors (CNIs) for septic patients in ICU: A cohort study from MIMIC database;2023-11-26
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