Melatonin-mediated inhibition of Cav3.2 T-type Ca2+channels induces sensory neuronal hypoexcitability through the novel protein kinase C-eta isoform

Author:

Zhang Yuan12,Ji Heyi1,Wang Jiangong1,Sun Yufang1,Qian Zhiyuan2,Jiang Xinghong1,Snutch Terrance P.3,Sun Yangang4,Tao Jin1ORCID

Affiliation:

1. Department of Physiology and Neurobiology & Centre for Ion Channelopathy; Medical College of Soochow University; Suzhou China

2. Department of Geriatrics & Institute of Neuroscience; the Second Affiliated Hospital of Soochow University; Suzhou China

3. Michael Smith Laboratories and Djavad Mowafaghian Centre for Brain Health; University of British Columbia; Vancouver BC Canada

4. Institute of Neuroscience; State Key Laboratory of Neuroscience; Shanghai Institutes for Biological Sciences; Chinese Academy of Sciences; Shanghai China

Funder

National Natural Science Foundation of China

Chinese Academy of Sciences

Government of Jiangsu Province

Jiangsu Six Talent Peaks Program

Priority Academic Program Development of Jiangsu Higher Education Institutions

Canadian Institutes of Health Research

Koerner Foundation and is the Canada Research Chair in Biotechnology and Genomics-Neurobiology

Publisher

Wiley

Subject

Endocrinology

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