Receptor for advanced glycation end products up‐regulation in cerebral endothelial cells mediates cerebrovascular‐related amyloid β accumulation after Porphyromonas gingivalis infection-Reference-Cited by-同舟云学术

Receptor for advanced glycation end products up‐regulation in cerebral endothelial cells mediates cerebrovascular‐related amyloid β accumulation after Porphyromonas gingivalis infection

Published:2020-06-15 Issue:3 Volume:158 Page:724-736
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Zeng Fan¹^ORCID,Liu Yicong²³,Huang Wanyi¹,Qing Hong⁴^ORCID,Kadowaki Tomoko⁵,Kashiwazaki Haruhiko⁶,Ni Junjun¹⁴^ORCID,Wu Zhou¹⁷^ORCID

Affiliation:

1. Department of Aging Science and Pharmacology Kyushu University Fukuoka Japan

2. The Affiliated Stomatology Hospital School of Medical Zhejiang University Zhejiang China

3. Key Laboratory of Oral Biomedical Research of Zhejiang Province School of Stomatology Zhejiang University Zhejiang China

4. Key Laboratory of Molecular Medicine and Biotherapy in the Ministry of Industry and Information Technology Department of Biology School of Life Science Beijing Institute of TechnologyHaidian District Beijing China

5. Division of Frontier Life Science Department of Medical and Dental Sciences Graduate School of Biomedical Sciences Nagasaki University Nagasaki Japan

6. Section of Geriatric Dentistry and Perioperative Medicine in Dentistry Division of Maxillofacial Diagnostic and Surgical Sciences Faculty of Dental Science Kyushu University Fukuoka Japan

7. Faculty of Dental Science OBT Research CenterKyushu University Fukuoka Japan

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jnc.15096

Reference50 articles.

1. Astrocyte–endothelial interactions at the blood–brain barrier

2. The overlap between vascular disease and Alzheimer’s disease - lessons from pathology

3. Peptidyl Arginine Deiminase from Porphyromonas gingivalis Abolishes Anaphylatoxin C5a Activity

4. Nuclear Factor-κB Mediates Up-Regulation of Cathepsin B by Doxorubicin in Tumor Cells

5. Blood-derived amyloid-β protein induces Alzheimer’s disease pathologies

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