S2′-subsite variations between human and mouse enzymes (plasmin, factor XIa, kallikrein) elucidate inhibition differences by tissue factor pathway inhibitor -2 domain1-wild-type, Leu17Arg-mutant and aprotinin

Author:

Vadivel K.1,Kumar Y.1,Ogueli G. I.1,Ponnuraj S. M.1,Wongkongkathep P.2,Loo J. A.23,Bajaj M. S.4,Bajaj S. P.13

Affiliation:

1. Department of Orthopaedic Surgery; University of California; Los Angeles CA USA

2. Department of Chemistry and Biochemistry; University of California; Los Angeles CA USA

3. Molecular Biology Institute; University of California; Los Angeles CA USA

4. Division of Pulmonology and Critical Care; Department of Medicine; University of California; Los Angeles CA USA

Funder

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Hematology

Reference75 articles.

1. Plasmin-mediated fibrinolysis by variant recombinant tissue plasminogen activators;Urano;Proc Natl Acad Sci USA,1989

2. The cell biology of the plasminogen system;Plow;FASEB J,1995

3. Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma;Kashuk;Ann Surg,2010

4. The incidence and magnitude of fibrinolytic activation in trauma patients;Raza;J Thromb Haemost,2013

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