Good response to infliximab in rheumatoid arthritis following failure of interleukin-1 receptor antagonist-Reference-Cited by-同舟云学术

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Good response to infliximab in rheumatoid arthritis following failure of interleukin-1 receptor antagonist

Published:2014-05-05 Issue:4 Volume:19 Page:370-376
ISSN:1756-1841
Container-title:International Journal of Rheumatic Diseases
language:en
Short-container-title:Int J Rheum Dis

Author:

Bao Jun¹²,Yue Tao³,Li Ting²,He Dong-Yi³,Bao Yi-Xiao¹

Affiliation:

1. Department of Pediatrics; Xinhua Hospital Shanghai Jiaotong University School of Medicine; Shanghai China

2. Department of Rheumatology & Immunology; Shanghai Changzheng Hospital; Second Military Medical University; Shanghai China

3. Department of Rheumatology; Shanghai Guanghua Hospital; Shanghai China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Rheumatology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/1756-185X.12387/fullpdf

Reference14 articles.

1. Tides of inflammation: impact of biologics;St. Clair;J Rheumatol,2002

2. American College of Rheumatology. Preliminary definition of improvement in rheumatoid arthritis;Felson;Arthritis Rheum,1995

3. Outcomes after switching from one anti-tumor necrosis factor α agent to a second anti-tumor necrosis factor α agent in patients with rheumatoid arthritis;Hyrich;Arthritis Rheum,2007

4. The American College of Rheumatology preliminary core set of disease activity measures for rheumatoid arthritis clinical trials;Felson;Arthritis Rheum,1993

5. Development and validation of the European League Against Rheumatism response criteria for rheumatoid arthritis: comparison with the preliminary American College of Rheumatology and the World Health Organization/International League Against Rheumatism criteria;Gestel;Arthritis Rheum,1996

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1. The Role of Biologics in Rheumatoid Arthritis: A Narrative Review;Cureus;2023-01-03

2. Identification of key candidate genes and biological pathways in the synovial tissue of patients with rheumatoid arthritis;Experimental and Therapeutic Medicine;2022-04-04

3. IL ‐35 ameliorates collagen‐induced arthritis by promoting TNF ‐α‐induced apoptosis of synovial fibroblasts and stimulating M2 macrophages polarization;The FEBS Journal;2019-03-23

4. Treatment Options for Refractory Kawasaki Disease: Alternative Treatments for Infliximab Nonresponders;Kawasaki Disease;2016-08-28

5. Interleukin-22 and rheumatoid arthritis: Emerging role in pathogenesis and therapy;Autoimmunity;2014-12-08

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