HNF4Amutation: switch from hyperinsulinaemic hypoglycaemia to maturity-onset diabetes of the young, and incretin response-Reference-Cited by-同舟云学术

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HNF4Amutation: switch from hyperinsulinaemic hypoglycaemia to maturity-onset diabetes of the young, and incretin response

Published:2014-02-16 Issue:3 Volume:31 Page:e11-e15
ISSN:0742-3071
Container-title:Diabetic Medicine
language:en
Short-container-title:Diabet. Med.

Author:

Arya V. B.¹²,Rahman S.²,Senniappan S.¹²,Flanagan S. E.³,Ellard S.³,Hussain K.¹²

Affiliation:

1. Department of Paediatric Endocrinology; Great Ormond Street Hospital for Children NHS; Trust London

2. The Institute of Child Health; University College London; Exeter UK

3. Institute of Biomedical and Clinical Science; University of Exeter Medical School; Exeter UK

Funder

Medical Council

Publisher

Wiley

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/dme.12369/fullpdf

Reference20 articles.

1. The different tissue transcription patterns of genes for HNF-1, C/EBP, HNF-3, and HNF-4, protein factors that govern liver-specific transcription;Xanthopoulos;Proc Natl Acad Sci U S A,1991

2. Liver-enriched transcription factor HNF-4 is a novel member of the steroid hormone receptor superfamily;Sladek;Genes Dev,1990

3. A transcriptional hierarchy involved in mammalian cell-type specification;Kuo;Nature,1992

4. Control of pancreas and liver gene expression by HNF transcription factors;Odom;Science,2004

5. Hepatocyte nuclear factor 4α regulates the expression of pancreatic β-cell genes implicated in glucose metabolism and nutrient-induced insulin secretion;Wang;J Biol Chem,2000

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