Downregulation but lack of promoter hypermethylation or somatic mutations of the potential tumor suppressorCXXC5in MDS and AML with deletion 5q
Author:
Affiliation:
1. Department of Hematology; Rigshospitalet; Copenhagen; Denmark
2. Karolinska University Hospital and Karolinska Institute; Stockholm; Sweden
3. Research and Innovation Centre (BRIC); University of Copenhagen; Copenhagen; Denmark
Publisher
Wiley
Subject
Hematology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/ejh.12045/fullpdf
Reference17 articles.
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2. Functional involvement of RINF, retinoid-inducible nuclear factor (CXXC5), in normal and tumoral human myelopoiesis;Pendino;Blood,2009
3. CXXC domain of human DNMT1 is essential for enzymatic activity;Pradhan;Biochemistry,2008
4. The MT domain of the proto-oncoprotein MLL binds to CpG-containing DNA and discriminates against methylation;Birke;Nucleic Acids Res,2002
5. Mbd1 is recruited to both methylated and nonmethylated CpGs via distinct DNA binding domains;Jørgensen;Mol Cell Biol,2004
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