High glucose protects cardiomyocytes against ischaemia/reperfusion injury by suppressing myocardiocyte apoptosis via circHIPK3/miR‐29b/AKT3 signalling

Author:

Cheng Nan1,Jin Chang2,Jin Ping3,Zhu Dan4,Hou Zuoxu5ORCID

Affiliation:

1. Department of Cardiovascular Surgery Chinese PLA General Hospital Beijing China

2. Medical Innovation Research Division Research Center for Biomedical Engineering Chinese PLA General Hospital Beijing China

3. Department of Cardiovascular Surgery The First Affiliated Hospital of Air Force Military Medical University Xi'an China

4. Department of Arrhythmia Qinghai Cardio‐Cerebrovascular Hospital Xi'ning China

5. Department of Thoracic Cardiovascular Surgery General Hospital of Central Theater Command of the People's Liberation Army Wuhan China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Reference37 articles.

1. Ischaemic conditioning and reperfusion injury;Hausenloy DJ;Nat Rev Cardiol,2016

2. The mechanisms and strategies to protect from hepatic ischemia‐reperfusion injury;Li J;Eur Rev Med Pharmacol Sci,2015

3. Oxidative stress‐mediated reperfusion injury: mechanism and therapies;Xia Z;Oxid Med Cell Longev,2014

4. Erythropoietin attenuates apoptosis after ischemia‐reperfusion‐induced renal injury in transiently hyperglycemic Wister rats;Caetano AM;Transplant Proc,2011

5. Evaluation of renal protection from high doses of melatonin in an experimental model of renal ischemia and reperfusion in hyperglycemic rats;Souza AV;Transplant Proc,2014

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