Spontaneous atopic dermatitis in mice with a defective skin barrier is independent of ILC2 and mediated by IL‐1β-Reference-Cited by-同舟云学术

Spontaneous atopic dermatitis in mice with a defective skin barrier is independent of ILC2 and mediated by IL‐1β

Published:2019-04-29 Issue:10 Volume:74 Page:1920-1933
ISSN:0105-4538
Container-title:Allergy
language:en
Short-container-title:Allergy

Author:

Schwartz Christian¹²^ORCID,Moran Tara¹³,Saunders Sean P.¹³^ORCID,Kaszlikowska Agnieszka¹³,Floudas Achilleas¹³^ORCID,Bom Joana⁴,Nunez Gabriel⁵,Iwakura Yoichiro⁶^ORCID,O’Neill Luke⁷^ORCID,Irvine Alan D.³⁸^ORCID,McKenzie Andrew N. J.⁹^ORCID,Ogg Graham¹⁰^ORCID,Walsh Patrick T.³¹¹^ORCID,Demengeot Jocelyne⁴^ORCID,Fallon Padraic G.¹³¹¹^ORCID

Affiliation:

1. School of Medicine Trinity Biomedical Sciences Institute, Trinity College Dublin Dublin Ireland

2. Mikrobiologisches Institut ‐ Klinische Mikrobiologie, Immunologie und Hygiene Universitätsklinikum Erlangen and Friedrich‐Alexander Universität (FAU) Erlangen‐Nürnberg Erlangen Germany

3. National Children’s Research Centre, Our Lady’s Children’s Hospital Dublin Ireland

4. Instituto Gulbenkian de Ciência Oeiras Portugal

5. Department of Pathology and Comprehensive Cancer Center University of Michigan Medical School Ann Arbor Michigan

6. Research Institute for Biomedical Sciences Tokyo University of Science Chiba Japan

7. School of Biochemistry and Immunology Trinity Biomedical Sciences Institute, Trinity College Dublin Dublin Ireland

8. Department of Paediatric Dermatology Our Lady’s Children’s Hospital Dublin Ireland

9. MRC Laboratory of Molecular Biology Cambridge UK

10. MRC Human Immunology Unit, Weatherall Institute of Molecular Medicine John Radcliffe Hospital University of Oxford Oxford UK

11. Trinity Translational Medicine Institute, St James’s Hospital, Trinity College Dublin Dublin Ireland

Funder

European Molecular Biology Organization

Science Foundation Ireland

Wellcome Trust

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/all.13801

Reference94 articles.

1. Atopic dermatitis

2. The interplay between genetic and environmental factors in the pathogenesis of atopic dermatitis

3. Atopic dermatitis phenotypes and the need for personalized medicine

4. Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis

5. Tmem79/Matt is the matted mouse gene and is a predisposing gene for atopic dermatitis in human subjects

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