The suppression of bromodomain and extra-terminal domain inhibits vascular inflammation by blocking NF-κB and MAPK activation
Author:
Affiliation:
1. Department of Rheumatology, The First Affiliated Hospital; Sun Yat-sen University; Guangzhou Guangdong China
2. Department of Rheumatology; The First People's Hospital of Foshan; Foshan Guangdong China
Funder
Guangdong Natural Science Foundation
National Natural Science Foundation of China
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.13657/fullpdf
Reference43 articles.
1. Endothelium as a therapeutic target in sepsis;Aird;Curr Drug Targets,2007
2. The Concise Guide to PHARMACOLOGY 2015/16: Enzymes;Alexander;Br J Pharmacol,2015
3. BET protein function is required for inflammation: Brd2 genetic disruption and BET inhibitor JQ1 impair mouse macrophage inflammatory responses;Belkina;J Immunol,2013
4. Alpha 4 integrins mediate lymphocyte attachment and rolling under physiologic flow;Berlin;Cell,1995
5. NF-kappaB directs dynamic super enhancer formation in inflammation and atherogenesis;Brown;Mol Cell,2014
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