CD36 is upregulated in mice with periodontitis and metabolic syndrome and involved in macrophage gene upregulation by palmitate

Author:

Lu Z1,Li Y1,Brinson CW1,Kirkwood KL2,Lopes-Virella MF13,Huang Y13

Affiliation:

1. Division of Endocrinology, Diabetes and Medical Genetics; Department of Medicine; College of Medicine; Medical University of South Carolina; Charleston SC USA

2. Department of Oral Health Science; College of Dental Medicine; Medical University of South Carolina; Charleston SC USA

3. Ralph H. Johnson Veterans Affairs Medical Center; Charleston SC USA

Funder

National Institutes of Health

Biomedical Laboratory Research and Development, VA Office of Research and Development

National Institute of General Medicine

Publisher

Wiley

Subject

General Dentistry,Otorhinolaryngology

Reference48 articles.

1. Melatonin improves glucose homeostasis in young Zucker diabetic fatty rats;Agil;J Pineal Res,2012

2. Metabolic syndrome a widespread threatening condition; risk factors, diagnostic criteria, therapeutic options, prevention and controversies: an overview;Amihaesei;Rev Med Chir Soc Med Nat Iasi,2014

3. Class B scavenger receptor types I and II and CD36 mediate bacterial recognition and proinflammatory signaling induced by Escherichia coli, lipopolysaccharide, and cytosolic chaperonin 60;Baranova;J Immunol,2012

4. Scavenger receptor CD36 mediates uptake of high density lipoproteins in mice and by cultured cells;Brundert;J Lipid Res,2011

5. Metabolic syndrome and periodontitis: is oxidative stress a common link?;Bullon;J Dent Res,2009

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