Proteinase‐activated receptor‐2 antagonist C391 inhibits Alternaria ‐induced airway epithelial signalling and asthma indicators in acute exposure mouse models-Reference-Cited by-同舟云学术

Proteinase‐activated receptor‐2 antagonist C391 inhibits Alternaria ‐induced airway epithelial signalling and asthma indicators in acute exposure mouse models

Published:2022-02 Issue: Volume: Page:
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:British J Pharmacology

Author:

Rivas Candy M.¹²,Yee Michael C.³,Addison Kenneth J.²⁴,Lovett Marissa¹,Pal Kasturi³,Ledford Julie G.²⁴,Dussor Gregory⁵,Price Theodore J.⁵,Vagner Josef⁶,DeFea Kathryn A.³⁷,Boitano Scott¹²⁴⁶⁸

Affiliation:

1. Physiological Sciences Graduate Interdisciplinary Program University of Arizona Tucson Arizona USA

2. Asthma and Airway Disease Research Center University of Arizona Health Sciences Tucson Arizona USA

3. Biomedical Sciences University of California Riverside Riverside California USA

4. Department of Cellular and Molecular Medicine University of Arizona Health Sciences Tucson Arizona USA

5. School of Behavioral and Brain Sciences and Center for Advanced Pain Studies University of Texas at Dallas Richardson Texas USA

6. Bio5 Collaborative Research Institute University of Arizona Tucson Arizona USA

7. Corporate Headquarters PARMedics, Inc. Temecula California USA

8. Department of Physiology University of Arizona Health Sciences Tucson Arizona USA

Funder

National Heart, Lung, and Blood Institute

National Institute of Allergy and Infectious Diseases

National Institute of Neurological Disorders and Stroke

Publisher

Wiley

Subject

Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bph.15745

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3. β‐Arrestin‐biased proteinase‐activated receptor‐2 antagonist C781 limits allergen‐induced airway hyperresponsiveness and inflammation;British Journal of Pharmacology;2022-12-05

4. Alternaria alternata-induced airway epithelial signaling and inflammatory responses via protease-activated receptor-2 expression;Biochemical and Biophysical Research Communications;2022-02