AttenuatedSalmonella VNP20009 mutant(ΔhtrA) is a promising candidate for bacteria-mediated tumour therapy in hosts with TNFR1 deficiency-Reference-Cited by-同舟云学术

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AttenuatedSalmonella VNP20009 mutant(ΔhtrA) is a promising candidate for bacteria-mediated tumour therapy in hosts with TNFR1 deficiency

Published:2018-05-24 Issue:1 Volume:67 Page:97-103
ISSN:0266-8254
Container-title:Letters in Applied Microbiology
language:en
Short-container-title:Lett Appl Microbiol

Author:

Xu W.¹,Zhou T.¹,Zhou J.¹,Qiang Z.¹,Zhang J.¹²^ORCID,Hua Z.¹²³

Affiliation:

1. The State Key Laboratory of Pharmaceutical Biotechnology; School of Life Sciences; Nanjing University; Nanjing China

2. Changzhou High-Tech Research Institute of Nanjing University and Jiangsu TargetPharma Laboratories Inc.; Changzhou China

3. Shenzhen Research Institute of Nanjing University; Shenzhen China

Funder

the Jiangsu Provincial Nature Science Foundation

the Chinese National Natural Sciences Foundation

the National Key R&D Research Program of China

Shenzhen Science and Technology Innovation Committee

Shenzhen Peacock Plan

Nanshan Distrcit Science and Technology Innovation Committee

Publisher

Wiley

Subject

Applied Microbiology and Biotechnology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/lam.12999/fullpdf

Reference20 articles.

1. A Salmonella typhimurium htrA live vaccine expressing multiple copies of a peptide comprising amino acids 8-24 of herpes simplex virus glycoprotein D as a genetic fusion to tetanus toxin fragment C protects mice from herpes simplex virus infection;Chabalgoity;Mol Microbiol,1996

2. Oral delivery of tumor-targeting Salmonella exhibits promising therapeutic efficacy and low toxicity;Chen;Cancer Sci,2009

3. Biodistribution and genetic stability of the novel antitumor agent VNP20009, a genetically modified strain of Salmonella typhimurium;Clairmont;J Infect Dis,2000

4. Susceptibility to Salmonella typhimurium infection and effectiveness of vaccination in mice deficient in the tumor necrosis factor alpha p55 receptor;Everest;Infect Immun,1998

5. Tumor necrosis factor-alpha is required in the protective immune response against Mycobacterium tuberculosis in mice;Flynn;Immunity,1995

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