A senescent cell bystander effect: senescence‐induced senescence
Author:
Affiliation:
1. Institute for Ageing and Health, Newcastle University, Campus for Ageing and Vitality, Newcastle upon Tyne NE4 5PL, UK
2. Department of Life Science, Walton Hall, Open University, Milton Keynes, MK7 6AA, UK
Publisher
Wiley
Subject
Cell Biology,Aging
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1474-9726.2012.00795.x
Reference20 articles.
1. Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders
2. The Gene Expression Program of Prostate Fibroblast Senescence Modulates Neoplastic Epithelial Cell Proliferation through Paracrine Mechanisms
3. Cellular senescence: when bad things happen to good cells
4. Up-regulation of ROS by mitochondria-dependent bystander signaling contributes to genotoxicity of bystander effects
5. Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor
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