p16INK4a-induced senescence is disabled by melanoma-associated mutations
Author:
Publisher
Wiley
Subject
Cell Biology,Aging
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1474-9726.2008.00422.x/fullpdf
Reference64 articles.
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2. G1 arrest by p16 (INK4A) uncouples growth from cell cycle progression in leukemia cells with deregulated cyclin E and c-Myc expression;Ausserlechner;Leukemia,2005
3. Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints;Bartkova;Nature,2006
4. Reversal of human cellular senescence: roles of the p53 and p16 pathways;Beausejour;EMBO J,2003
5. Functional impairment of melanoma-associated p16INK4a mutants in melanoma cells despite retention of cyclin-dependent kinase 4 binding;Becker;Clin. Cancer Res,2001
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