The telomeric protein SNM1B/Apollo is required for normal cell proliferation and embryonic development
Author:
Publisher
Wiley
Subject
Cell Biology,Aging
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1474-9726.2010.00631.x/fullpdf
Reference52 articles.
1. Dynamic dependence on ATR and ATM for double-strand break repair in human embryonic stem cells and neural descendants;Adams;PLoS ONE,2010
2. Snm1-deficient mice exhibit accelerated tumorigenesis and susceptibility to infection;Ahkter;Mol. Cell. Biol.,2005
3. SNM1A acts downstream of ATM to promote the G1 cell cycle checkpoint;Akhter;Biochem. Biophys. Res. Commun.,2008
4. Deficiency in SNM1 abolishes an early mitotic checkpoint induced by spindle stress;Akhter;Mol. Cell. Biol.,2004
5. Snm1B/Apollo mediates replication fork collapse and S Phase checkpoint activation in response to DNA interstrand cross-links;Bae;Oncogene,2008
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1. DNA-PK and the TRF2 iDDR inhibit MRN-initiated resection at leading-end telomeres;Nature Structural & Molecular Biology;2023-08-31
2. DCLRE1B/hSNM1B (Apollo) is not acutely required for human pluripotent stem cell survival;2023-07-30
3. DNA-PK and the TRF2 iDDR inhibit MRN-initiated resection at leading-end telomeres;2023-03-06
4. Genetics of human telomere biology disorders;Nature Reviews Genetics;2022-09-23
5. Dual targeting of Saccharomyces cerevisiae Pso2 to mitochondria and the nucleus, and its functional relevance in the repair of DNA interstrand crosslinks;G3 Genes|Genomes|Genetics;2022-04-28
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