Effects of ketone bodies in Alzheimer's disease in relation to neural hypometabolism, β-amyloid toxicity, and astrocyte function-Reference-Cited by-同舟云学术

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Effects of ketone bodies in Alzheimer's disease in relation to neural hypometabolism, β-amyloid toxicity, and astrocyte function

Published:2015-04-23 Issue:1 Volume:134 Page:7-20
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Hertz Leif¹,Chen Ye²,Waagepetersen Helle S.³

Affiliation:

1. Laboratory of Metabolic Brain Diseases; Institute of Metabolic Disease Research and Drug Development; China Medical University; Shenyang China

2. Henry M. Jackson Foundation; Bethesda Maryland USA

3. Department of Drug Design and Pharmacology; Faculty of Health and Medical Sciences; University of Copenhagen; Copenhagen Denmark

Funder

University of Copenhagen

Danish Council for Independent Research: Medical Sciences

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.13107/fullpdf

Reference161 articles.

1. Activation of PARP by oxidative stress induced by beta-amyloid: implications for Alzheimer's disease;Abeti;Neurochem. Res.,2012

2. Beta-amyloid activates PARP causing astrocytic metabolic failure and neuronal death;Abeti;Brain,2011

3. Changes in intracellular calcium and glutathione in astrocytes as the primary mechanism of amyloid neurotoxicity;Abramov;J. Neurosci.,2003

4. Mitochondrial dysfunction and Alzheimer's disease;Albrekkan;Open J. Endocr. Metab. Dis.,2013

5. Triheptanoin supplementation to ketogenic diet curbs cognitive impairment in APP/PS1 mice used as a model of familial Alzheimer's disease;Aso;Curr. Alzheimer Res.,2013

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