Tumor‐like features of gene expression and metabolic profiles in enlarged pancreatic islets are associated with impaired incretin‐induced insulin secretion in obese diabetes: A study of Zucker fatty diabetes mellitus rat-Reference-Cited by-同舟云学术

Tumor‐like features of gene expression and metabolic profiles in enlarged pancreatic islets are associated with impaired incretin‐induced insulin secretion in obese diabetes: A study of Zucker fatty diabetes mellitus rat

Published:2020-05-29 Issue:6 Volume:11 Page:1434-1447
ISSN:2040-1116
Container-title:Journal of Diabetes Investigation
language:en
Short-container-title:J. Diabetes Investig.

Author:

Hayami Tomohide¹²³^ORCID,Yokoi Norihide¹²^ORCID,Yamaguchi Takuro¹²,Honda Kohei¹,Murao Naoya¹,Takahashi Harumi¹²,Wang Shujie⁴,Seino Yusuke⁵^ORCID,Kamiya Hideki³,Yabe Daisuke¹²^ORCID,Sweet Ian R⁶,Mizoguchi Akira⁴,Nakamura Jiro³,Seino Susumu¹²^ORCID

Affiliation:

1. Division of Molecular and Metabolic Medicine Department of Physiology and Cell Biology Kobe University Graduate School of Medicine Kobe Japan

2. Kansai Electric Power Medical Research Institute Kobe Japan

3. Division of Diabetes Department of Internal Medicine Aichi Medical University Nagakute Japan

4. Department of Neural Regeneration and Cell Communication Mie University Graduate School of Medicine Tsu Japan

5. Division of Endocrinology and Metabolism Department of Internal Medicine Fujita Health University Toyoake Japan

6. Department of Medicine Division of Metabolism, Endocrinology and Nutrition University of Washington Seattle Washington USA

Funder

Japan Agency for Medical Research and Development

Japan Society for the Promotion of Science

Publisher

Wiley

Subject

General Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jdi.13272

Reference45 articles.

1. Incretin Action in the Pancreas: Potential Promise, Possible Perils, and Pathological Pitfalls

2. Reduced incretin effect in Type 2 (non-insulin-dependent) diabetes

3. Obesity may attenuate the HbA1c-lowering effect of sitagliptin in Japanese type 2 diabetic patients

4. Incretin physiology and pathophysiology from an A sian perspective

5. Glutamate Acts as a Key Signal Linking Glucose Metabolism to Incretin/cAMP Action to Amplify Insulin Secretion

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