Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia-reperfusion in a rat model-Reference-Cited by-同舟云学术

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Arginase inhibition improves coronary microvascular function and reduces infarct size following ischaemia-reperfusion in a rat model

Published:2013-04-15 Issue:2 Volume:208 Page:172-179
ISSN:1748-1708
Container-title:Acta Physiologica
language:en
Short-container-title:Acta Physiol

Author:

Grönros J.¹,Kiss A.¹,Palmér M.²,Jung C.¹,Berkowitz D.³,Pernow J.¹

Affiliation:

1. Division of Cardiology; Department of Medicine; Karolinska Institute; Karolinska University Hospital; Stockholm; Sweden

2. Department of Bioscience; Astra Zeneca R&D; Mölndal; Sweden

3. Johns Hopkins University School of Medicine; Baltimore; MD; USA

Publisher

Wiley

Subject

Physiology

Reference25 articles.

1. Reperfusion injury: experimental evidence and clinical implications;Ambrosio;Am Heart J,1999

2. Arginase reciprocally regulates nitric oxide synthase activity and contributes to endothelial dysfunction in aging blood vessels;Berkowitz;Circulation,2003

3. Nitric oxide is a preconditioning mimetic and cardioprotectant and is the basis of many available infarct-sparing strategies;Cohen;Cardiovasc Res,2006

4. Adenosine and cardioprotection during reperfusion-an overview;Donato;Mol Cell Biochem,2003

5. Predictors and long-term prognosis of angiographic slow/no-reflow phenomenon during emergency percutaneous coronary intervention for ST-elevated acute myocardial infarction;Dong-bao;Clin Cardiol,2010

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