PKM2 promotes angiotensin‐II‐induced cardiac remodelling by activating TGF‐β/Smad2/3 and Jak2/Stat3 pathways through oxidative stress-Reference-Cited by-同舟云学术

PKM2 promotes angiotensin‐II‐induced cardiac remodelling by activating TGF‐β/Smad2/3 and Jak2/Stat3 pathways through oxidative stress

Published:2021-10-23 Issue:22 Volume:25 Page:10711-10723
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Zhang Xiyu¹,Zheng Cuiting¹,Gao Zhenqiang¹,Wang Lingling²,Chen Chen³,Zheng Yuanyuan⁴,Meng Yan¹^ORCID

Affiliation:

1. Beijing Key Laboratory of Metabolic Disorders Related Cardiovascular Diseases Beijing Lab for Cardiovascular Precision Medicine Department of Pathology Capital Medical University Beijing China

2. Department of Pathology Beijing Shijitan Hospital Capital Medical University Beijing China

3. China‐America Institute of Neuroscience Beijing Luhe Hospital Capital Medical University Beijing China

4. Department of Pharmacology Capital Medical University Beijing China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.17007

Reference45 articles.

1. Regression of Established Cardiac Fibrosis in Hypertensive Heart Disease

2. Cardiac remodeling at the population level—risk factors, screening, and outcomes

3. Inhibition of Hypertrophy Is a Good Therapeutic Strategy in Ventricular Pressure Overload

4. EphrinB2 Regulates Cardiac Fibrosis Through Modulating the Interaction of Stat3 and TGF-β/Smad3 Signaling

5. Transforming growth factor-β receptor antagonism attenuates myocardial fibrosis in mice with cardiac-restricted overexpression of tumor necrosis factor

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