The rs553668 polymorphism of theADRA2Agene predicts the worsening of fasting glucose values in a cohort of subjects without diabetes. A population-based study
Author:
Publisher
Wiley
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1464-5491.2011.03522.x/fullpdf
Reference8 articles.
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2. Transgenic mice overexpressing α2a adrenoceptors in pancreatic β-cells show altered regulation of glucose homeostasis;Devedjian;Diabetologia,2000
3. Overexpression of α2A-adrenergic receptors contributes to type 2 diabetes;Rosengren;Science,2010
4. Variants at DGKB/TMEM195, ADRA2A, GLIS3 and C2CD4B loci are associated with reduced glucose-stimulated β-cell function in middle-aged Danish people;Boesgaard;Diabetologia,2010
5. Genetic predisposition to long-term non-diabetic deteriorations in glucose homeostasis: ten-year follow-up of the Glacier Study;Renström;Diabetes,2011
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3. The ADRA2A rs553668 variant is associated with type 2 diabetes and five variants were associated at nominal significance levels in a population-based case–control study from Mexico City;Gene;2018-08
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