Increased all-cause and cardiovascular mortality in monogenic diabetes as a result of mutations in the HNF1A gene
Author:
Publisher
Wiley
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1464-5491.2009.02913.x/fullpdf
Reference19 articles.
1. Defining the genetic aetiology of monogenic diabetes can improve treatment;Gloyn;Expert Opin,2006
2. Different genes, different diabetes: lessons from maturity-onset diabetes of the young;Stride;Ann Med,2002
3. Altered insulin secretory responses to glucose in diabetic and non-diabetic subjects with mutations in the diabetes susceptibility gene MODY3 on chromosome 12;Byrne;Diabetes,1996
4. Beta-cell genes and diabetes: quantitative and qualitative differences in the pathophysiology of hepatic nuclear factor-1α and glucokinase mutations;Pearson;Diabetes,2001
5. Sensitivity to sulphonylureas in patients with hepatocyte nuclear factor-1α gene mutations: evidence for pharmacogenetics in diabetes;Pearson;Diabet Med,2005
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4. Diabetic Ketoacidosis in Patients with Maturity-Onset Diabetes of the Young;Experimental and Clinical Endocrinology & Diabetes;2024-06-05
5. Diabetische Ketoazidose bei einer Patientin mit HNF4A-MODY;Diabetologie und Stoffwechsel;2024-02-27
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