Paradoxical HDL-C reduction during rosiglitazone and fibrate treatment
Author:
Publisher
Wiley
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1464-5491.2007.02029.x/fullpdf
Reference17 articles.
1. Risk factors for coronary artery disease in non-insulin dependent diabetes mellitus: United Kingdom Prospective Diabetes Study (UKPDS;Turner;Br Med J,1998
2. Rosiglitazone monotherapy is effective in patients with Type 2 diabetes;Lebovitz;J Clin Endocrinol Metab,2001
3. Effects of pioglitazone and rosiglitazone on blood lipid levels and glycemic control in patients with type 2 diabetes mellitus: a retrospective review of randomly selected medical records;Boyle;Clin Ther,2002
4. A meta-analysis comparing the effect of thiazolidinediones on cardiovascular risk factors;Chiquette;Arch Intern Med,2004
5. Paradoxical high-density lipoprotein reduction induced by fenofibrate and ciprofibrate;Collinson;Ann Clin Biochem,1996
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1. Targeted Proteomics Identifies Paraoxonase/Arylesterase 1 (PON1) and Apolipoprotein Cs as Potential Risk Factors for Hypoalphalipoproteinemia in Diabetic Subjects Treated with Fenofibrate and Rosiglitazone;Molecular & Cellular Proteomics;2016-03
2. Use of fibrates in the metabolic syndrome: A review;World Journal of Diabetes;2016
3. Metabolic syndrome: A review of the role of vitamin D in mediating susceptibility and outcome;World Journal of Diabetes;2015
4. Treatment of metabolic syndrome with ankaflavin, a secondary metabolite isolated from the edible fungus Monascus spp.;Applied Microbiology and Biotechnology;2014-04-13
5. Paradoxical Reduction in HDL-C With Fenofibrate and Thiazolidinedione Therapy in Type 2 Diabetes: The ACCORD Lipid Trial;Diabetes Care;2014-02-11
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