Essential role ofBordetellaNadC in a quinolinate salvage pathway for NAD biosynthesis-Reference-Cited by-同舟云学术

Essential role ofBordetellaNadC in a quinolinate salvage pathway for NAD biosynthesis

Published:2016-11-25 Issue:3 Volume:103 Page:423-438
ISSN:0950-382X
Container-title:Molecular Microbiology
language:en
Short-container-title:Molecular Microbiology

Author:

Brickman Timothy J.¹,Suhadolc Ryan J.¹,McKelvey Pamela J.¹,Armstrong Sandra K.¹

Affiliation:

1. Department of Microbiology and ImmunologyUniversity of Minnesota Medical School, 3‐117 Microbiology Research Facility689 23^rd Ave. S.EMinneapolis MN55455‐1507 USA

Funder

University of Minnesota Faculty Incentive Research Account

Publisher

Wiley

Subject

Molecular Biology,Microbiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/mmi.13566

Reference72 articles.

1. NAD+Released during Inflammation Participates in T Cell Homeostasis by Inducing ART2-Mediated Death of Naive T Cells In Vivo

2. Overrepresentation of a Gene Family Encoding Extracytoplasmic Solute Receptors in Bordetella

3. Roles for Cationic Residues at the Quinolinic Acid Binding Site of Quinolinate Phosphoribosyltransferase

4. Interactions at the 2 and 5 Positions of 5-Phosphoribosyl Pyrophosphate Are Essential in Salmonella typhimurium Quinolinate Phosphoribosyltransferase

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3. Bacterial NadQ (COG4111) is a Nudix-like, ATP-responsive regulator of NAD biosynthesis;Journal of Structural Biology;2022-12

4. Cyclic di-GMP Regulates the Type III Secretion System and Virulence in Bordetella bronchiseptica;Infection and Immunity;2022-06-16

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