The α5 Neuronal Nicotinic Acetylcholine Receptor Subunit Plays an Important Role in the Sedative Effects of Ethanol But Does Not Modulate Consumption in Mice

Author:

Santos Nathan1,Chatterjee Susmita1,Henry Andrea1,Holgate Joan2,Bartlett Selena E.

Affiliation:

1. Ernest Gallo Clinic and Research Center; University of California, San Francisco; Emeryville; California

2. Translational Research Institute ; Queensland University of Technology; Brisbane; Queensland; Australia

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Reference50 articles.

1. Ethanol modulation of nicotinic acetylcholine receptor currents in cultured cortical neurons;Aistrup;Mol Pharmacol,1999

2. Antagonism of ethanol ataxia by intracerebellar nicotine: possible modulation by mouse cerebellar nitric oxide and cGMP;Al-Rejaie;Brain Res Bull,2006

3. The mesolimbic dopamine-activating properties of ethanol are antagonized by mecamylamine;Blomqvist;Eur J Pharmacol,1993

4. Ethanol-induced locomotor activity: involvement of central nicotinic acetylcholine receptors?;Blomqvist;Brain Res Bull,1992

5. Effects of ethanol on recombinant human neuronal nicotinic acetylcholine receptors expressed in Xenopus oocytes;Cardoso;J Pharmacol Exp Ther,1999

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