High homocysteine levels prevent via H 2 S the CoCl 2 ‐induced alteration of lymphocyte viability-Reference-Cited by-同舟云学术

High homocysteine levels prevent via H 2 S the CoCl 2 ‐induced alteration of lymphocyte viability

Published:2016-04-08 Issue:8 Volume:20 Page:1411-1419
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Bruzzese Laurie¹,Fenouillet Emmanuel¹²,Fromonot Julien¹³,Durand‐Gorde Josée‐Martine¹,Condo Jocelyne¹,Kipson Nathalie¹,Mottola Giovanna¹³,Deharo Pierre¹⁴,Guieu Régis¹³,Ruf Jean¹⁵

Affiliation:

1. UMR‐MD2 Marseille Medical School Aix‐Marseille University and IRBA Northern sector Marseille France

2. CNRS Institut des Sciences Biologiques France

3. Laboratory of Biochemistry Timone University Hospital Marseille France

4. Cardiology Department Timone University Hospital Marseille France

5. INSERM France

Funder

Aix-Marseille Université

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.12829

Reference51 articles.

1. Mechanisms of Cardiovascular Remodeling in Hyperhomocysteinemia

2. Increased levels of C‐reactive protein and interleukin‐6 in hyperhomocysteinemic subjects

3. Homocysteine at pathophysiologic concentrations activates human monocyte and induces cytokine expression and inhibits macrophage migration inhibitory factor expression

4. A proinflammatory state is associated with hyperhomocysteinemia in the elderly

5. Potential role for adenosine in the pathogenesis of the vascular complications of hyperhomocysteinemia

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