Fluorofenidone attenuates pulmonary inflammation and fibrosis via inhibiting the activation of NALP 3 inflammasome and IL ‐1β/ IL ‐1R1/MyD88/ NF ‐κB pathway-Reference-Cited by-同舟云学术

Fluorofenidone attenuates pulmonary inflammation and fibrosis via inhibiting the activation of NALP 3 inflammasome and IL ‐1β/ IL ‐1R1/MyD88/ NF ‐κB pathway

Published:2016-06-16 Issue:11 Volume:20 Page:2064-2077
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Song Cheng¹²,He Lujuan¹,Zhang Jin³,Ma Hong³,Yuan Xiangning³,Hu Gaoyun⁴,Tao Lijian³,Zhang Jian⁵,Meng Jie¹

Affiliation:

1. Department of Respiratory Medicine Xiangya Hospital Central South University Changsha China

2. Department of Respiratory Medicine Central Hospital of Wuhan Tongji Medical College Huazhong University of Science & Technology Wuhan China

3. Department of Nephrology Medicine Xiangya Hospital Central South University Changsha China

4. Pharmaceutical School Central South University Changsha China

5. Department of Microbial Infection & Immunity The Ohio State University Columbus OH USA

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.12898

Reference34 articles.

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2. Idiopathic Pulmonary Fibrosis: Prevailing and Evolving Hypotheses about Its Pathogenesis and Implications for Therapy

3. Mechanisms and Mediators of Pulmonary Fibrosis

4. Sputum matrix metalloproteinase-9, tissue inhibitor of metalloprotinease-1, and their molar ratio in patients with chronic obstructive pulmonary disease, idiopathic pulmonary fibrosis and healthy subjects

5. The absence of reactive oxygen species production protects mice against bleomycin-induced pulmonary fibrosis

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