Clinical and immunological phenotype switch to prurigo nodularis in a patient receiving ixekizumab for treating psoriasis: a case report
Author:
Affiliation:
1. Department of Dermatology The First Affiliated Hospital, Zhejiang University School of Medicine Hangzhou People's Republic of China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Dermatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/ijd.16658
Reference5 articles.
1. The IL-23/IL-17 Pathway in Inflammatory Skin Diseases: From Bench to Bedside
2. Clinicopathologic overlap of psoriasis, eczema, and psoriasiform dermatoses: A retrospective study of T helper type 2 and 17 subsets, interleukin 36, and β-defensin 2 in spongiotic psoriasiform dermatitis, sebopsoriasis, and tumor necrosis factor α inhibitor–associated dermatitis
3. Prurigo Nodularis Is Characterized by Systemic and Cutaneous T Helper 22 Immune Polarization
4. Analysis of anti-tumour necrosis factor-induced skin lesions reveals strong T helper 1 activation with some distinct immunological characteristics
5. Early tissue responses in psoriasis to the antitumour necrosis factor-α biologic etanercept suggest reduced interleukin-17 receptor expression and signalling
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1. Ixekizumab/methotrexate;Reactions Weekly;2023-10-28
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