Physiological aspects of Toll-like receptor 4 activation in sepsis-induced acute kidney injury-Reference-Cited by-同舟云学术

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Physiological aspects of Toll-like receptor 4 activation in sepsis-induced acute kidney injury

Published:2016-10-08 Issue:3 Volume:219 Page:575-590
ISSN:1748-1708
Container-title:Acta Physiologica
language:en
Short-container-title:Acta Physiol

Author:

Anderberg S. B.¹,Luther T.¹,Frithiof R.¹

Affiliation:

1. Department of Surgical Sciences; Section of Anesthesia & Intensive Care; Uppsala University; Uppsala Sweden

Funder

Statens Naturvidenskabelige Forskningsrad

Deutsche Forschungsgemeinschaft

Vetenskapsrådet

Publisher

Wiley

Subject

Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/apha.12798/fullpdf

Reference129 articles.

1. The hematologic system as a marker of organ dysfunction in sepsis;Aird;Mayo Clin Proc,2003

2. Histones from dying renal cells aggravate kidney injury via TLR2 and TLR4;Allam;J Am Soc Nephrol,2012

3. Cutting edge: Tlr5−/− mice are more susceptible to Escherichia coli urinary tract infection;Andersen-Nissen;J Immunol,2007

4. Severe sepsis and septic shock;Angus;N Engl J Med,2013

5. Cyclooxygenase 2 inhibition suppresses tubuloglomerular feedback: roles of thromboxane receptors and nitric oxide;Araujo;Am J Physiol Renal Physiol,2009

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