Potential therapeutic targeting of inflammation in epidermolysis bullosa simplex
Author:
Affiliation:
1. St John's Institute of Dermatology Guy's and St Thomas' NHS Foundation Trust London SE1 7EH U.K.
Publisher
Wiley
Subject
Dermatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjd.17106
Reference11 articles.
1. Point mutations in human keratin 14 genes of epidermolysis bullosa simplex patients: Genetic and functional analyses
2. A mutation in the conserved helix termination peptide of keratin 5 in hereditary skin blistering
3. Cytokines as genetic modifiers in K5���/���mice and in human epidermolysis bullosa simplex
4. Keratin 1 maintains skin integrity and participates in an inflammatory network in skin via interleukin-18
5. An Autocrine/Paracrine Loop Linking Keratin 14 Aggregates to Tumor Necrosis Factor α-mediated Cytotoxicity in a Keratinocyte Model of Epidermolysis Bullosa Simplex
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1. Pathological Mechanisms Involved in Epidermolysis Bullosa Simplex: Current Knowledge and Therapeutic Perspectives;International Journal of Molecular Sciences;2024-08-31
2. Keratins as an Inflammation Trigger Point in Epidermolysis Bullosa Simplex;International Journal of Molecular Sciences;2021-11-18
3. Epidermolysis bullosa simplex clearance after nasopharyngeal carcinoma treatment;JAAD Case Reports;2021-06
4. A Novel Mutation p.L461P in KRT5 Causing Localized Epidermolysis Bullosa Simplex;Annals of Dermatology;2021
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