Diffuse Midline Gliomas with Histone H3-K27M Mutation: A Series of 47 Cases Assessing the Spectrum of Morphologic Variation and Associated Genetic Alterations
Author:
Affiliation:
1. Division of Neuropathology, Department of Pathology; University of California; San Francisco CA
2. Department of Neurological Surgery; University of California
3. Department of Pediatrics; University of California
Funder
UCSF Brain Tumor SPORE and an NIH Director's Early Independence Award
Publisher
Wiley
Subject
Clinical Neurology,Pathology and Forensic Medicine,General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/bpa.12336/fullpdf
Reference31 articles.
1. H3F3A K27M mutations in thalamic gliomas from young adult patients;Aihara;Neuro Oncol,2014
2. Specific detection of methionine 27 mutation in histone 3 variants (H3K27M) in fixed tissue from high-grade astrocytomas;Bechet;Acta Neuropathol,2014
3. Reduced H3K27me3 and DNA hypomethylation are major drivers of gene expression in K27M mutant pediatric high-grade gliomas;Bender;Cancer Cell,2013
4. Clinical, radiological, histological and molecular characteristics of paediatric epithelioid glioblastoma;Broniscer;Neuropathol Appl Neurobiol,2014
5. Histopathologic spectrum of paediatric diffuse intrinsic pontine glioma: diagnostic and therapeutic implications;Buczkowicz;Acta Neuropathol,2014
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