Pathophysiology of pachyonychia congenita‐associated palmoplantar keratoderma: new insights into skin epithelial homeostasis and avenues for treatment-Reference-Cited by-同舟云学术

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Pathophysiology of pachyonychia congenita‐associated palmoplantar keratoderma: new insights into skin epithelial homeostasis and avenues for treatment

Published:2019-07-24 Issue:3 Volume:182 Page:564-573
ISSN:0007-0963
Container-title:British Journal of Dermatology
language:en
Short-container-title:Br J Dermatol

Author:

Zieman A.G.¹^ORCID,Coulombe P.A.¹²

Affiliation:

1. Department of Cell and Developmental Biology University of Michigan Medical School 3071 Biomedical Sciences Research Building, 109 Zina Pitcher Place Ann Arbor MI 48109 U.S.A.

2. Department of Dermatology University of Michigan Medical School 3071 Biomedical Sciences Research Building, 109 Zina Pitcher Place Ann Arbor MI 48109 U.S.A.

Funder

National Institute of Arthritis and Musculoskeletal and Skin Diseases

National Cancer Institute

Publisher

Wiley

Subject

Dermatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjd.18033

Reference111 articles.

1. Clinical and Pathological Features of Pachyonychia Congenita

2. Keratin K6c Mutations Cause Focal Palmoplantar Keratoderma

3. Keratin 16 and keratin 17 mutations cause pachyonychia congenita

4. A Mutation in Human Keratin K6b Produces a Phenocopy of the K17 Disorder Pachyonychia Congenita Type 2

5. Mutation of a type II keratin gene (K6a) in pachyonychia congenita

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