Notch signaling pathway in keloid disease: Enhanced fibroblast activity in a Jagged-1 peptide-dependent manner in lesional vs. extralesional fibroblasts-Reference-Cited by-同舟云学术

Notch signaling pathway in keloid disease: Enhanced fibroblast activity in a Jagged-1 peptide-dependent manner in lesional vs. extralesional fibroblasts

Published:2012-09 Issue:5 Volume:20 Page:688-706
ISSN:1067-1927
Container-title:Wound Repair and Regeneration
language:en
Short-container-title:Wound Repair Regen

Author:

Syed Farhatullah,Bayat Ardeshir

Publisher

Wiley

Subject

Dermatology,Surgery

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1524-475X.2012.00823.x/fullpdf

Reference39 articles.

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2. Fibroblasts from the growing margin of keloid scars produce higher levels of collagen I and III compared with intralesional and extralesional sites: clinical implications for lesional site-directed therapy;Syed;Br J Dermatol,2010

3. Tumor-like stem cells derived from human keloid are governed by the inflammatory niche driven by IL-17/IL-6 axis;Zhang;PLoS ONE,2009

4. Genetics of keloid scarring;Shih;Arch Dermatol Res,2010

5. Fibronectin is overproduced by keloid fibroblasts during abnormal wound healing;Babu;Mol Cell Biol,1989

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