Reactive oxygen species‐mediated endoplasmic reticulum stress response induces apoptosis ofMycobacterium avium‐infected macrophages by activating regulated IRE1‐dependent decay pathway-Reference-Cited by-同舟云学术

Reactive oxygen species‐mediated endoplasmic reticulum stress response induces apoptosis ofMycobacterium avium‐infected macrophages by activating regulated IRE1‐dependent decay pathway

Published:2019-08-16 Issue:12 Volume:21 Page:
ISSN:1462-5814
Container-title:Cellular Microbiology
language:en
Short-container-title:Cellular Microbiology

Author:

Go Dam¹²,Lee Junghwan¹²,Choi Ji‐Ae¹²,Cho Soo‐Na¹²,Kim Seon‐Hwa¹²,Son Sang‐Hun¹²,Song Chang‐Hwa¹²³^ORCID

Affiliation:

1. Department of Medical ScienceChungnam National University Daejeon South Korea

2. Department of Microbiology, College of MedicineChungnam National University Daejeon South Korea

3. Research Institute for Medical Sciences, College of MedicineChungnam National University Daejeon South Korea

Funder

Chungnam National University

Publisher

Hindawi Limited

Subject

Virology,Immunology,Microbiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cmi.13094

Reference33 articles.

1. Diagnosis and treatment of lung infection with nontuberculous mycobacteria

2. Apoptosis is an innate defense function of macrophages against Mycobacterium tuberculosis

3. Execution of Macrophage Apoptosis by Mycobacterium avium through Apoptosis Signal-regulating Kinase 1/p38 Mitogen-activated Protein Kinase Signaling and Caspase 8 Activation

4. Endoplasmic reticulum stress response is involved inMycobacterium tuberculosisprotein ESAT-6-mediated apoptosis

5. Mycobacterial HBHA induces endoplasmic reticulum stress-mediated apoptosis through the generation of reactive oxygen species and cytosolic Ca2+ in murine macrophage RAW 264.7 cells

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