Hepatic Inactivation of the Type 2 Deiodinase Confers Resistance to Alcoholic Liver Steatosis-Reference-Cited by-同舟云学术

Hepatic Inactivation of the Type 2 Deiodinase Confers Resistance to Alcoholic Liver Steatosis

Published:2019-04-26 Issue:7 Volume:43 Page:1376-1383
ISSN:0145-6008
Container-title:Alcoholism: Clinical and Experimental Research
language:en
Short-container-title:Alcohol Clin Exp Re

Author:

Fonseca Tatiana L.¹,Fernandes Gustavo W.¹,Bocco Barbara M. L. C.¹,Keshavarzian Ali²,Jakate Shriram³,Donohue Terrence M.⁴,Gereben Balázs⁵,Bianco Antonio C.¹^ORCID

Affiliation:

1. Section of Endocrinology, Diabetes and Metabolism University of Chicago Chicago Illinois

2. Division of Digestive Diseases and Nutrition Rush University Chicago Illinois

3. Department of Pathology Rush University Chicago Illinois

4. Department of Internal Medicine University of Nebraska Medical Center Omaha Nebraska

5. Department of Endocrine Neurobiology Institute of Experimental Medicine Hungarian Academy of Sciences Budapest Hungary

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acer.14027

Reference30 articles.

1. Mouse model of chronic and binge ethanol feeding (the NIAAA model)

2. Chronic ingestion of ethanol stimulates lipogenic response in rat hepatocytes

3. Disruption of Thyroid Hormone Activation in Type 2 Deiodinase Knockout Mice Causes Obesity With Glucose Intolerance and Liver Steatosis Only at Thermoneutrality

4. Targeting Aldehyde Dehydrogenase 2: New Therapeutic Opportunities

5. Histologic Findings in Alcoholic Liver Disease

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