Fibroblast growth factor 21 attenuates the progression of hyperuricemic nephropathy through inhibiting inflammation, fibrosis and oxidative stress-Reference-Cited by-同舟云学术

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Fibroblast growth factor 21 attenuates the progression of hyperuricemic nephropathy through inhibiting inflammation, fibrosis and oxidative stress

Published:2022-09-28 Issue:6 Volume:131 Page:474-486
ISSN:1742-7835
Container-title:Basic & Clinical Pharmacology & Toxicology
language:en
Short-container-title:Basic Clin Pharma Tox

Author:

Jiang Xinghao¹,Wu Qing¹,Opoku Yeboah Kwaku¹²,Zou Yimeng¹,Wang Dan¹,Hu Changhui¹,Ren Guiping¹³^ORCID

Affiliation:

1. Biopharmaceutic Lab, College of Life Science Northeast Agricultural University Harbin China

2. Department of Biology Education, Faculty of Science Education University of Education Winneba Ghana

3. Key Laboratory of Agricultural Biological Function Gene Northeast Agricultural University Harbin China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Pharmacology,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bcpt.13793

Reference36 articles.

1. The genetic basis of hyperuricaemia and gout

2. Research Advances in the Mechanisms of Hyperuricemia-Induced Renal Injury

3. Soluble Uric Acid Activates the NLRP3 Inflammasome

4. Pharmacological inhibition of autophagy by 3-MA attenuates hyperuricemic nephropathy

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Fibroblast growth factor 21 mitigates lupus nephritis progression via the FGF21/Irgm 1/NLRP3 inflammasome pathway;International Immunopharmacology;2024-04

2. FGF21 Inhibits Hypoxia/Reoxygenation-induced Renal Tubular Epithelial Cell Injury by Regulating the PPARγ/NF-κB Signaling Pathway;Cell Biochemistry and Biophysics;2024-03-08

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