Suppression of androgen receptor signaling induces prostate cancer migration via activation of the <scp>CCL20</scp> – <scp>CCR6</scp> axis-Reference-Cited by-同舟云学术

Suppression of androgen receptor signaling induces prostate cancer migration via activation of the CCL20 – CCR6 axis

Published:2022-12-18 Issue:4 Volume:114 Page:1479-1490
ISSN:1347-9032
Container-title:Cancer Science
language:en
Short-container-title:Cancer Science

Author:

Kano Hiroshi¹,Izumi Kouji¹^ORCID,Hiratsuka Kaoru¹,Toriumi Ren¹^ORCID,Nakagawa Ryunosuke¹^ORCID,Aoyama Shuhei¹,Kamijima Taiki¹,Shimada Takafumi¹,Naito Renato¹^ORCID,Kadomoto Suguru¹^ORCID,Iwamoto Hiroaki¹^ORCID,Yaegashi Hiroshi¹^ORCID,Kawaguchi Shohei¹^ORCID,Nohara Takahiro¹,Shigehara Kazuyoshi¹,Kadono Yoshifumi¹,Saito Yohei²,Nakagawa‐Goto Kyoko²,Yoshioka Kazuaki³,Nakata Hiroki¹⁴,Lin Wen‐Jye⁵,Mizokami Atsushi¹

Affiliation:

1. Department of Integrative Cancer Therapy and Urology Kanazawa University Graduate School of Medical Science Kanazawa Japan

2. School of Pharmaceutical Sciences, College of Medical, Pharmaceutical and Health Science Kanazawa University Kanazawa Japan

3. Department of Vascular Physiology Kanazawa University Graduate School of Medical Science Kanazawa Japan

4. Department of Clinical Engineering, Faculty of Health Sciences Komatsu University Komatsu Japan

5. Immunology Research Center National Health Research Institutes Miaoli County Taiwan

Publisher

Wiley

Subject

Cancer Research,Oncology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cas.15683

Reference36 articles.

1. Cancer Statistics, 2021

2. Prostate Cancer, Version 2.2019, NCCN Clinical Practice Guidelines in Oncology

3. Androgen receptor signaling‐targeted therapy and taxane chemotherapy induce visceral metastasis in castration‐resistant prostate cancer

4. Targeting the androgen receptor with siRNA promotes prostate cancer metastasis through enhanced macrophage recruitment via CCL2/CCR2‐induced STAT3 activation

5. C-C motif ligand 5 promotes migration of prostate cancer cells in the prostate cancer bone metastasis microenvironment

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