Subsaturation of the N‐methyl‐D‐aspartate receptor glycine site allows the regulation of bursting activity in juvenile rat nigral dopamine neurons
Author:
Affiliation:
1. GIGA‐Neurosciences Neurophysiology Group University of Liege Liege Belgium
Publisher
Wiley
Subject
General Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/ejn.14491
Reference91 articles.
1. Immunohistochemical localization of N-methyl-d-aspartate and α-amino-3-hydroxy-5-methyl-4-isoxazolepropionate receptor subunits in the substantia nigra pars compacta of the rat
2. Severe Impairment of NMDA Receptor Function in Mice Carrying Targeted Point Mutations in the Glycine Binding Site Results in Drug-Resistant Nonhabituating Hyperactivity
3. Glycine Uptake Governs Glycine Site Occupancy at NMDA Receptors of Excitatory Synapses
4. Modulation of N-methyl-D-aspartate receptor function by glycine transport
5. Active release of glycine or d-serine saturates the glycine site of NMDA receptors at the cerebellar mossy fibre to granule cell synapse
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