Episodic ataxia type 1 mutations in theKCNA1gene impair the fast inactivation properties of the human potassium channels Kv1.4-1.1/Kvβ1.1 and Kv1.4-1.1/Kvβ1.2-Reference-Cited by-同舟云学术

Episodic ataxia type 1 mutations in theKCNA1gene impair the fast inactivation properties of the human potassium channels Kv1.4-1.1/Kvβ1.1 and Kv1.4-1.1/Kvβ1.2

Published:2006-12 Issue:11 Volume:24 Page:3073-3083
ISSN:0953-816X
Container-title:European Journal of Neuroscience
language:en
Short-container-title:

Author:

Imbrici Paola,D'Adamo Maria Cristina,Kullmann Dimitri M.,Pessia Mauro

Publisher

Wiley

Subject

General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1460-9568.2006.05186.x/fullpdf

Reference59 articles.

1. Episodic ataxia results from voltage-dependent potassium channels with altered functions;Adelman;Neuron,1995

2. Inactivation and recovery in Kv1.4 K+ channels: lipophilic interactions at the intracellular mouth of the pore;Bett;J. Physiol.,2004

3. Control of human potassium channel inactivation by editing of a small mRNA hairpin;Bhalla;Nat. Struct. Mol. Biol.,2004

4. Episodic ataxia/myokymia syndrome is associated with point mutations in the human potassium channel gene, KCNA1 [see comments];Browne;Nat. Genet.,1994

5. Novel form of spreading acidification and depression in the cerebellar cortex demonstrated by neutral red optical imaging;Chen;J. Neurophysiol.,1999

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