Enhanced extinction of cocaine seeking in brain-derived neurotrophic factor Val66Met knock-in mice
Author:
Publisher
Wiley
Subject
General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1460-9568.2012.08021.x/fullpdf
Reference69 articles.
1. The BDNF Val66Met polymorphism affects HPA-axis reactivity to acute stress;Alexander;Psychoneuroendocrinology,2010
2. The brain-derived neurotrophic factor Val66Met polymorphism is associated with reduced functional magnetic resonance imaging activity in the hippocampus and increased use of caudate nucleus-dependent strategies in a human virtual navigation task;Banner;Eur. J. Neurosci.,2011
3. Gene expression profiling of facilitated L-LTP in VP16-CREB mice reveals that BDNF is critical for the maintenance of LTP and its synaptic capture;Barco;Neuron,2005
4. A BDNF infusion into the medial prefrontal cortex suppresses cocaine seeking in rats;Berglind;Eur. J. Neurosci.,2007
5. A role for alpha-adrenergic receptors in extinction of conditioned fear and cocaine conditioned place preference;Bernardi;Behav. Neurosci.,2010
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2. Association Between Brain-Derived Neurotrophic Factor Val66Met Polymorphism and Methamphetamine Use Disorder: A Meta-Analysis;Frontiers in Psychiatry;2020-11-19
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4. Mice engineered to mimic a common Val66Met polymorphism in the BDNF gene show greater sensitivity to reversal in environmental contingencies;Developmental Cognitive Neuroscience;2018-11
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