Excitotoxic death induced by released glutamate in depolarized primary cultures of mouse cerebellar granule cells is dependent on GABAAreceptors and niflumic acid-sensitive chloride channels-Reference-Cited by-同舟云学术

Excitotoxic death induced by released glutamate in depolarized primary cultures of mouse cerebellar granule cells is dependent on GABAAreceptors and niflumic acid-sensitive chloride channels

Published:2005-01 Issue:1 Volume:21 Page:103-112
ISSN:0953-816X
Container-title:European Journal of Neuroscience
language:en
Short-container-title:

Author:

Babot Zoila,Cristòfol Rosa,Suñol Cristina

Publisher

Wiley

Subject

General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1460-9568.2004.03848.x/fullpdf

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3. Na-K-Cl cotransporter contributes to glutamate-mediated excitotoxicity;Beck;J. Neurosci.,2003

4. Domoic acid neurotoxicity in cultured cerebellar granule neurons is mediated predominantly by NMDA receptors that are activated as a consequence of excitatory amino acid release;Berman;J. Neurochem.,1997

5. Brevetoxins cause acute excitotoxicity in primary cultures of rat cerebellar granule neurons;Berman;J. Pharmacol. Exp. Ther.,1999

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