Differential Effects of an L-type Ca2+Channel Antagonist on Activity- and Phosphorylation-enhanced Release of Acetylcholine at the Neuromuscular Junction of the FrogIn Vitro-Reference-Cited by-同舟云学术

Differential Effects of an L-type Ca2+Channel Antagonist on Activity- and Phosphorylation-enhanced Release of Acetylcholine at the Neuromuscular Junction of the FrogIn Vitro

Published:1996-03 Issue:3 Volume:8 Page:437-445
ISSN:0953-816X
Container-title:European Journal of Neuroscience
language:en
Short-container-title:

Author:

Arenson M. S.,Gill D. S.

Publisher

Wiley

Subject

General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1460-9568.1996.tb01227.x/fullpdf

Reference57 articles.

1. Protein phosphatase inhibitor okadaic acid enhances transmitter release at neuromuscular junctions;Abdul-Ghani;Proc. Natl Acad. Sci. USA,1991

2. Calcium channel effectors are potent non-competitive blockers of acetylcholine receptors;Adam;Pflügers Arch.,1990

3. A possible involvement of cyclic AMP in the expression of desensitization of the nicotinic acetylcholine receptor;Albuquerque;FEBS Lett.,1986

4. ω-Conotoxin does not block the verapamil-sensitive calcium channels at mouse motor nerve terminals;Anderson;Neurosci. Lett.,1987

5. The effects of muscarine and atropine reveal that inhibitory autoreceptors are present on frog motor nerve terminals but are not activated during transmission;Arenson;Naunyn-Schmiedeberg's Arch. Pharmacol.,1991

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