A multi‐hit hypothesis for an APOE4 ‐dependent pathophysiological state
Author:
Affiliation:
1. School of Life Sciences University of Sussex Brighton UK
2. School of Psychology University of Sussex Brighton UK
Funder
Alzheimer's Society
Publisher
Wiley
Subject
General Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/ejn.15685
Reference341 articles.
1. Human Apolipoprotein E ε4 Expression Impairs Cerebral Vascularization and Blood—Brain Barrier Function in Mice
2. Apolipoprotein E4 Causes Age- and Tau-Dependent Impairment of GABAergic Interneurons, Leading to Learning and Memory Deficits in Mice
3. APOE4 is Associated with Differential Regional Vulnerability to Bioenergetic Deficits in Aged APOE Mice
4. Impaired cerebral glucose metabolism and cognitive functioning predict deterioration in mild cognitive impairment
5. Sex and APOE ε4 genotype modify the Alzheimer’s disease serum metabolome
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